Restless legs syndrome

Triggered by a complex patient with myasthenia, polyneuropathy, RLS, organic psychosis and several more we discuss what is to be known about the restless legs syndrome, using

For me the first source provided a fundamental insight into the pathophysiology of RLS (heightened circadianic dopamine variations with consecutive desensitization of dopamine receptors) that finally explains why dopaminergic medication can lead to augmentation.

I still can’t understand why we use long-acting dopamine agonists rather than short-acting or at least SR L-Dopa to cover symptoms and this pathophysiologic view would support the view.

Another brilliant development is transcranial sonography for imaging of the SN, finding hypoechogenicity – as reported in

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