After the recent introduction of Trobalt into the antiepileptic armamentarium it is time to talk about it’s pharmacology, potential and current applications. We won’t speculate about all standard avenues that are open to new AEDs (pain, anxiety, depression, bipolar disease, withdrawal, restless legs, migraine) until clinical studies have been either faked (Gabapentin), refute these applications or (rarely) establish them as additional indications. We neither intend to speculate on the neuroprotective effect every AED initially was supposed to have had.

We use the following references:


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